Neuronal ApoE4 stimulates C/EBPβ activation, promoting Alzheimer’s disease pathology in a mouse model

This publication has 72 references indexed in Scilit:

• The influence of Apolipoprotein E genotype on regional pathology in Alzheimer’s disease
  BMC Neurology, 2013
• Plasma Signaling Proteins in Persons at Genetic Risk for Alzheimer Disease
  Archives of Neurology, 2012
• ApoE-Directed Therapeutics Rapidly Clear β-Amyloid and Reverse Deficits in AD Mouse Models
  Science, 2012
• Apolipoprotein E isoforms and regulation of the innate immune response in brain of patients with Alzheimer's disease
  Current Opinion in Neurobiology, 2011
• Similarities and differences in structure, expression, and functions of VLDLR and ApoER2
  Molecular Neurodegeneration, 2011
• Actions of β-Amyloid Protein on Human Neurons Are Expressed through the Amylin Receptor
  The American Journal of Pathology, 2011
• Decreased Clearance of CNS β-Amyloid in Alzheimer’s Disease
  Science, 2010
• Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease
  Nature Genetics, 2009
• The Role of Apolipoprotein E in Alzheimer's Disease
  Neuron, 2009
• Neuroprotective Actions of PIKE-L by Inhibition of SET Proteolytic Degradation by Asparagine Endopeptidase
  Molecular Cell, 2008