Treponema denticola, Porphyromonas gingivalis, and Tannerella forsythia induce cell death and release of endogenous danger signals
- 1 January 2017
- journal article
- Published by Elsevier in Archives of Oral Biology
- Vol. 73, 72-78
Abstract
No abstract availableKeywords
Funding Information
- Basic Science Research Program through the National Research Foundation of Korea
- Ministry of Science
- ICT & Future Planning (2014R1A1A3052018)
This publication has 36 references indexed in Scilit:
- Discovery of potential serum protein biomarkers for lymph node metastasis in oral cancerHead & Neck, 2015
- Correlation of Toll‐Like Receptor 4, Interleukin‐18, Transaminases, and Uric Acid in Patients With Chronic Periodontitis and Healthy AdultsJournal of Periodontology, 2015
- HMGB1 Is a Therapeutic Target for Sterile Inflammation and InfectionAnnual Review of Immunology, 2011
- The Inflammasome NLRs in Immunity, Inflammation, and Associated DiseasesAnnual Review of Immunology, 2011
- Pocket Epithelium in the Pathological Setting for HMGB1 ReleaseJournal of Dental Research, 2010
- Sterile inflammation: sensing and reacting to damageNature Reviews Immunology, 2010
- Virulence Factors of the Oral Spirochete Treponema denticolaJournal of Dental Research, 2010
- HMGB1 loves companyJournal of Leukocyte Biology, 2009
- Pyroptosis: host cell death and inflammationNature Reviews Microbiology, 2009
- Endogenous ligands of Toll-like receptors: implications for regulating inflammatory and immune responsesTrends in Immunology, 2002