Selective, Reversible Caspase-3 Inhibitor Is Neuroprotective and Reveals Distinct Pathways of Cell Death after Neonatal Hypoxic-ischemic Brain Injury
Authors:
Open Access
- 1 August 2002
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 277 (33) , 30128-30136
Abstract
No abstract availableKeywords
This publication has 64 references indexed in Scilit:
- Early Neurodegeneration after Hypoxia-Ischemia in Neonatal Rat Is Necrosis while Delayed Neuronal Death Is ApoptosisNeurobiology of Disease, 2001
- Clusterin contributes to caspase-3–independent brain injury following neonatal hypoxia-ischemiaNature Medicine, 2001
- Implication of cysteine proteases calpain, cathepsin and caspase in ischemic neuronal death of primatesProgress in Neurobiology, 2000
- BDNF Blocks Caspase-3 Activation in Neonatal Hypoxia–IschemiaNeurobiology of Disease, 2000
- Caspase inhibitor affords neuroprotection with delayed administration in a rat model of neonatal hypoxic-ischemic brain injury.Journal of Clinical Investigation, 1998
- Experimental Biology of Cerebral Hypoxia-Ischemia: Relation to Perinatal Brain DamagePediatric Research, 1990
- MK-801 prevents hypobaric-ischemic neuronal degeneration in infant rat brainJournal of Neuroscience, 1989
- Selective sparing of NADPH‐diaphorase neurons in neonatal hypoxia‐ischemiaAnnals of Neurology, 1988
- Neurotransmitter alterations in a model of perinatal hypoxic‐ischemic brain injuryAnnals of Neurology, 1983
- The influence of immaturity on hypoxic‐ischemic brain damage in the ratAnnals of Neurology, 1981