Obesity-Induced Coronary Microvascular Disease Is Prevented by iNOS Deletion and Reversed by iNOS Inhibition

This publication has 48 references indexed in Scilit:

• Impairment of Coronary Arteriolar Endothelium-Dependent Dilation after Multi-Walled Carbon Nanotube Inhalation: A Time-Course Study
  International Journal of Molecular Sciences, 2012
• Cell-penetrating nitroxides as molecular sensors for imaging of cancer in vivo , based on tissue redox activity
  Molecular BioSystems, 2012
• Characterization of the Thoracodorsal Artery: Morphology and Reactivity
  Microcirculation, 2012
• Displacement-encoded and manganese-enhanced cardiac MRI reveal that nNOS, not eNOS, plays a dominant role in modulating contraction and calcium influx in the mammalian heart
  American Journal of Physiology-Heart and Circulatory Physiology, 2012
• Vascular Generation of Tumor Necrosis Factor-α Reduces Nitric Oxide Availability in Small Arteries From Visceral Fat of Obese Patients
  JACC, 2011
• Design and validation of Segment - freely available software for cardiovascular image analysis
  BMC Medical Imaging, 2010
• Endothelial and vascular dysfunctions and insulin resistance in rats fed a high-fat, high-sucrose diet
  American Journal of Physiology-Heart and Circulatory Physiology, 2008
• Obesity induces a phenotypic switch in adipose tissue macrophage polarization
  Journal of Clinical Investigation, 2007
• Mechanism of vasodilation to adenosine in coronary arterioles from patients with heart disease
  American Journal of Physiology-Heart and Circulatory Physiology, 2005
• Isoflurane-induced Dilation of Porcine Coronary Arterioles Is Mediated by ATP-sensitive Potassium Channels
  Anesthesiology, 1998