Toll-like receptor-3-induced mitochondrial dysfunction in cultured human hepatocytes
Authors:
- Madhusudanarao VudaMVMadhusudanarao Vuda
- Jukka TakalaJTJukka Takala
- Matthias OchsMOMatthias Ochs
- Stephan M. JakobSJStephan M. Jakob
- 1 January 2011
- journal article
- Published by Elsevier in Mitochondrion
- Vol. 11 (1) , 83-88
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Astrocytes recognize intracellular polyinosinic‐polycytidylic acid via MDA‐5The FASEB Journal, 2008
- TLR3 is an endogenous sensor of tissue necrosis during acute inflammatory eventsJournal of Experimental Medicine, 2008
- Rapid increase in hospitalization and mortality rates for severe sepsis in the United States: A trend analysis from 1993 to 2003*Critical Care Medicine, 2007
- Role of caspase‐8 in hepatocyte response to infection and injury in mice†Hepatology, 2007
- ACTIVATION OF COMMON ANTIVIRAL PATHWAYS CAN POTENTIATE INFLAMMATORY RESPONSES TO SEPTIC SHOCKShock, 2006
- Bench-to-bedside review: Mechanisms of critical illness – classifying microcirculatory flow abnormalities in distributive shockCritical Care, 2006
- Cutting Edge: Innate Immunity Conferred by B Cells Is Regulated by Caspase-8The Journal of Immunology, 2005
- Association between mitochondrial dysfunction and severity and outcome of septic shockThe Lancet, 2002
- Recognition of double-stranded RNA and activation of NF-κB by Toll-like receptor 3Nature, 2001
- Epidemiology of severe sepsis in the United States: Analysis of incidence, outcome, and associated costs of careCritical Care Medicine, 2001