The LTB4-BLT1 Axis Mediates Neutrophil Infiltration and Secondary Injury in Experimental Spinal Cord Injury
- 1 May 2010
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 176 (5) , 2352-2366
Abstract
No abstract availableKeywords
Funding Information
- General Insurance Association of Japan
- Takeda Science Foundation
- Astellas Foundation for Research on Metabolic Disorders
- Japan Orthopaedics and Traumatology Foundation (0180)
This publication has 58 references indexed in Scilit:
- MMP-9–Positive Neutrophil Infiltration Is Associated to Blood–Brain Barrier Breakdown and Basal Lamina Type IV Collagen Degradation During Hemorrhagic Transformation After Human Ischemic StrokeStroke, 2008
- Inflammation and its role in neuroprotection, axonal regeneration and functional recovery after spinal cord injuryExperimental Neurology, 2008
- TNF-α BLOCKAGE IN A MOUSE MODEL OF SCIShock, 2008
- Complement Plays an Important Role in Spinal Cord Injury and Represents a Therapeutic Target for Improving Recovery following TraumaThe American Journal of Pathology, 2006
- Leukotriene B4 Receptor and the Function of Its Helix 8Journal of Biological Chemistry, 2005
- Extracellular signal-regulated kinase-mediated IL-1-induced cortical neuron damage during traumatic brain injuryNeuroscience Letters, 2005
- Delivery of hyper‐interleukin‐6 to the injured spinal cord increases neutrophil and macrophage infiltration and inhibits axonal growthJournal of Comparative Neurology, 2002
- Caspases determine the vulnerability of oligodendrocytes in the ischemic brainJournal of Clinical Investigation, 2000
- Leukotrienes promote plasma leakage and leukocyte adhesion in postcapillary venules: in vivo effects with relevance to the acute inflammatory response.Proceedings of the National Academy of Sciences, 1981
- Leukotriene B, a potent chemokinetic and aggregating substance released from polymorphonuclear leukocytesNature, 1980