TNF-α stimulates the expression of RANK during orthodontic tooth movement
Authors:
- 1 September 2020
- journal article
- research article
- Published by Elsevier in Archives of Oral Biology
- Vol. 117, 104796
Abstract
No abstract availableKeywords
Funding Information
- Japan Society for the Promotion of Science (16K11776)
- Japan Society for the Promotion of Science (18K09862)
- Japan Society for the Promotion of Science (19K10397)
This publication has 23 references indexed in Scilit:
- IL-4 inhibits TNF-α-mediated osteoclast formation by inhibition of RANKL expression in TNF-α-activated stromal cells and direct inhibition of TNF-α-activated osteoclast precursors via a T-cell-independent mechanism in vivoBone, 2012
- Anti-c-Fms antibody inhibits lipopolysaccharide-induced osteoclastogenesis in vivoFEMS Immunology & Medical Microbiology, 2011
- Osteoclasts: What Do They Do and How Do They Do It?The American Journal of Pathology, 2007
- RANK ligandThe International Journal of Biochemistry & Cell Biology, 2007
- RANKL–RANK signaling in osteoclastogenesis and bone diseaseTrends in Molecular Medicine, 2006
- TNF-α induces osteoclastogenesis by direct stimulation of macrophages exposed to permissive levels of RANK ligandJournal of Clinical Investigation, 2000
- Bone Resorption by OsteoclastsScience, 2000
- Identification and Characterization of the New Osteoclast Progenitor with Macrophage Phenotypes Being Able to Differentiate into Mature OsteoclastsJournal of Bone and Mineral Research, 2000
- Tumor Necrosis Factor α Stimulates Osteoclast Differentiation by a Mechanism Independent of the Odf/Rankl–Rank InteractionJournal of Experimental Medicine, 2000
- Orthodontic forces increase tumor necrosis factor α in the human gingival sulcusAmerican Journal of Orthodontics and Dentofacial Orthopedics, 1995