Clinicopathological features and mutational landscape of colorectal cancer subgroups defined by MSI and EMAST status
- 24 November 2025
- journal article
- Published by Elsevier in Pathology - Research and Practice
- Vol. 277, 156302
Abstract
No abstract availableKeywords
Funding Information
- Croatian Science Foundation
This publication has 33 references indexed in Scilit:
- Exploring the role of sporadic BRAF and KRAS mutations during colorectal cancer pathogenesis: A spotlight on the contribution of the endosome-lysosome systemCancer Letters, 2024
- Regulation of KRAS protein expression by miR-544a and KRAS-LCS6 polymorphism in wild-type KRAS sporadic colon adenocarcinomaHuman Cell, 2021
- Elevated Microsatellite Alterations at Selected Tetranucleotides (EMAST) Is Not Attributed to MSH3 Loss in Stage I-III Colon cancer: An Automated, Digitalized Assessment by Immunohistochemistry of Whole Slides and Hot SpotsTranslational Oncology, 2019
- Patterns of germline and somatic mutations in 16 genes associated with mismatch repair function or containing tandem repeat sequencesCancer Medicine, 2019
- Deciphering Elevated Microsatellite Alterations at Selected Tetra/Pentanucleotide Repeats, Microsatellite Instability, and Loss of Heterozygosity in Colorectal CancersThe Journal of Molecular Diagnostics, 2018
- Elevated microsatellite alterations at selected tetranucleotides in early‐stage colorectal cancers with and without high‐frequency microsatellite instability: same, same but different?Cancer Medicine, 2016
- Microsatellite Instability, EMAST, and Morphology Associations with T Cell Infiltration in Colorectal NeoplasiaDigestive Diseases and Sciences, 2011
- The landscape of somatic copy-number alteration across human cancersNature, 2010
- Histopathological Identification of Colon Cancer with Microsatellite InstabilityThe American Journal of Pathology, 2001
- A genetic model for colorectal tumorigenesisCell, 1990