Diplatin, a novel water-soluble platinum complex, inhibits lung cancer growth via augmentation of Fas-mediated apoptosis
Authors:
- 1 July 2020
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 879, 173128
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (81803544)
- Wenzhou Municipal Science and Technology Bureau (Y20160525)
This publication has 27 references indexed in Scilit:
- Diplatin, a Novel and Low-Toxicity Anti-Lung Cancer Platinum Complex, Activation of Cell Death in Tumors via a ROS/JNK/p53-Dependent Pathway, and a Low Rate of Acquired Treatment ResistanceFrontiers in Pharmacology, 2019
- Effects of Early Integrated Palliative Care on Caregivers of Patients with Lung and Gastrointestinal Cancer: A Randomized Clinical TrialThe Oncologist, 2017
- Cisplatin resistance and opportunities for precision medicinePharmacological Research, 2016
- Tacrolimus and the Risk of Solid Cancers After Liver Transplant: A Dose Effect RelationshipAmerican Journal of Transplantation, 2015
- Glaucocalyxin A Activates FasL and Induces Apoptosis Through Activation of the JNK Pathway in Human Breast Cancer CellsAsian Pacific Journal of Cancer Prevention, 2013
- Highly water-soluble platinum(ii) complexes as GLUT substrates for targeted therapy: improved anticancer efficacy and transporter-mediated cytotoxic propertiesChemical Communications, 2013
- Molecular mechanisms of cisplatin resistance in bladder cancerExpert Review of Anticancer Therapy, 2012
- Lung Cancer: Epidemiology, Etiology, and PreventionClinics in Chest Medicine, 2011
- Cisplatin: a review of toxicities and therapeutic applicationsVeterinary and Comparative Oncology, 2008
- Long‐term activation of SAPK/JNK, p38 kinase and fas‐L expression by cisplatin is attenuated in human carcinoma cells that acquired drug resistanceInternational Journal of Cancer, 2004