Mutations of the Ki-ras protooncogene in 3-methylcholanthrene and urethan-induced and butylated hydroxytoluene-promoted lung tumors of strain A/J and SWR mice
Authors:
- 4 May 1995
- journal article
- Published by Elsevier in Cancer Letters
- Vol. 91 (1) , 33-39
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Ki‐ras oncogene mutations in tumors and DNA adducts formed by benz[j]aceanthrylene and benzo[a]pyrene in the lungs of strain A/J miceMolecular Carcinogenesis, 1993
- Comparison of Ki-ras gene mutation among simultaneously occurring multiple urethan-induced lung tumors in individual miceCarcinogenesis, 1992
- Strain A/J Mouse Lung Adenoma Growth Patterns Vary When Induced by Different CarcinogensToxicologic Pathology, 1991
- Metabolism of the potent carcinogen 3-methylcholanthrylene by rat liver microsomesCarcinogenesis, 1990
- Neutron radiation can activate K-ras via a point mutation in codon 146 and induces a different spectrum of ras mutations than does gamma radiation.Molecular and Cellular Biology, 1990
- The ras gene family and human carcinogenesisMutation Research/Reviews in Genetic Toxicology, 1988
- Oncogene Activation in Chemical Carcinogenesis1988
- ras GENESAnnual Review of Biochemistry, 1987
- Biological and biochemical properties of human rasH genes mutated at codon 61Cell, 1986
- The metabolic activation and nucleic acid adducts of naturally-occurring carcinogens: Recent results with ethyl carbamate and the spice flavors safrole and estragoleBritish Journal of Cancer, 1983