Increased phospholipase A2 and thromboxane but not prostacyclin production by placental trophoblast cells from normal and preeclamptic pregnancies cultured under hypoxia condition
Authors:
- Yuping WangYWYuping Wang
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Impaired Oxygen-dependent Reduction of HIF-1α and -2α Proteins in Pre-eclamptic PlacentaePlacenta, 2003
- Hypoxia Attenuates PGE2but Increases Prostacyclin and Thromboxane Production in Human Term Villous TrophoblastPlacenta, 2001
- Increased Superoxide Generation is Associated with Decreased Superoxide Dismutase Activity and mRNA Expression in Placental Trophoblast Cells in Pre-eclampsiaPlacenta, 2001
- Oxygen and Placental Development During the First Trimester: Implications for the Pathophysiology of Pre-eclampsiaPlacenta, 2000
- Hypoxia limits differentiation and up-regulates expression and activity of prostaglandin H synthase 2 in cultured trophoblast from term human placentaAmerican Journal of Obstetrics and Gynecology, 1999
- Regulation of Human Placental Development by Oxygen TensionScience, 1997
- Placental Cytokines and the Pathogenesis of PreeclampsiaAmerican Journal of Reproductive Immunology, 1997
- Hypoxia alters early gestation human cytotrophoblast differentiation/invasion in vitro and models the placental defects that occur in preeclampsia.Journal of Clinical Investigation, 1996
- Placental lipid peroxides and thromboxane are increased and prostacyclin is decreased in women with preeclampsiaAmerican Journal of Obstetrics and Gynecology, 1992
- Control of vascular resistance in the human placentaPlacenta, 1992