Relevance of transgenic mouse models for Alzheimer's disease

This publication has 54 references indexed in Scilit:

• N-truncated amyloid β (Aβ) 4-42 forms stable aggregates and induces acute and long-lasting behavioral deficits
  Acta Neuropathologica, 2013
• Mouse models of Alzheimer's disease
  Brain Research Bulletin, 2012
• Pyroglutamate Amyloid β (Aβ) Aggravates Behavioral Deficits in Transgenic Amyloid Mouse Model for Alzheimer Disease
  Journal of Biological Chemistry, 2012
• Abnormal Cognition, Sleep, EEG and Brain Metabolism in a Novel Knock-In Alzheimer Mouse, PLB1
  PLOS ONE, 2011
• Pathogenic Forms of Tau Inhibit Kinesin-Dependent Axonal Transport through a Mechanism Involving Activation of Axonal Phosphotransferases
  The Journal of Neuroscience, 2011
• Aβ42‐driven cerebral amyloidosis in transgenic mice reveals early and robust pathology
  EMBO Reports, 2006
• The Arctic Alzheimer mutation facilitates early intraneuronal Aβ aggregation and senile plaque formation in transgenic mice
  Neurobiology of Aging, 2006
• Persistent Amyloidosis following Suppression of Aβ Production in a Transgenic Model of Alzheimer Disease
  PLOS Medicine, 2005
• Role of presenilin-1 in cortical lamination and survival of Cajal-Retzius neurons
  Developmental Biology, 2005
• A vector for expressing foreign genes in the brains and hearts of transgenic mice
  Genetic Analysis: Biomolecular Engineering, 1996