sFlt-1-enriched exosomes induced endothelial cell dysfunction and a preeclampsia-like phenotype in mice
Open Access
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- BM mesenchymal stromal cell–derived exosomes facilitate multiple myeloma progressionJournal of Clinical Investigation, 2013
- Angiogenic Factors and the Risk of Adverse Outcomes in Women With Suspected PreeclampsiaCirculation, 2012
- Transcriptionally Active Syncytial Aggregates in the Maternal Circulation May Contribute to Circulating Soluble Fms-Like Tyrosine Kinase 1 in PreeclampsiaHypertension, 2012
- Glyceryl Trinitrate Inhibits Hypoxia-Induced Release of Soluble fms-Like Tyrosine Kinase-1 and Endoglin from Placental TissuesThe American Journal of Pathology, 2011
- Role of Endothelin in Mediating Soluble fms-Like Tyrosine Kinase 1–Induced Hypertension in Pregnant RatsHypertension, 2010
- Hypoxia Enhances FGF2- and VEGF-Stimulated Human Placental Artery Endothelial Cell Proliferation: Roles of MEK1/2/ERK1/2 and PI3K/AKT1 PathwaysPlacenta, 2009
- The VEGF receptor Flt-1 spatially modulates Flk-1 signaling and blood vessel branchingJournal of Cell Biology, 2008
- Angiotensin II Induces Soluble fms-Like Tyrosine Kinase-1 Release via Calcineurin Signaling Pathway in PregnancyCirculation Research, 2007
- PreeclampsiaHypertension, 2005
- Circulating Angiogenic Factors and the Risk of PreeclampsiaNew England Journal of Medicine, 2004