Helicobacter pylori infection: Physiopathologic implication of Nα-methyl histamine

This publication has 32 references indexed in Scilit:

• Suppression of Helicobacter pylori reduces gastrin releasing peptide stimulated gastrin release in duodenal ulcer patients.
  Gut, 1992
• Helicobacter pylori infection induces a decrease in immunoreactive-somatostatin concentrations of human stomach
  Digestive Diseases and Sciences, 1992
• Helicobacter pyloriand Peptic Ulcer Disease
  New England Journal of Medicine, 1991
• Characterization and tissue distribution of H3 histamine receptors in guinea pigs by Nα-methylhistamine
  Biochemical and Biophysical Research Communications, 1990
• Antral Helicobacter pylori, hypergastrinaemia, and duodenal ulcers: effect of eradicating the organism.
  BMJ, 1989
• Simultaneous determination of histamine andNα-Methylhistamine in biological samples by an improved enzymatic single isotope assay
  Inflammation Research, 1985
• Auto-inhibition of brain histamine release mediated by a novel class (H3) of histamine receptor
  Nature, 1983
• A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
  Analytical Biochemistry, 1976
• Relationship between the inhibition constant (KI) and the concentration of inhibitor which causes 50 per cent inhibition (I50) of an enzymatic reaction
  Biochemical Pharmacology, 1973
• THE ROLE OF HISTAMINE IN GASTRIC HYDROCHLORIC ACID SECRETION
  Annals of the New York Academy of Sciences, 1962