Glycolysis-Independent Glucose Metabolism Distinguishes TE from ICM Fate during Mammalian Embryogenesis
- 19 March 2020
- journal article
- research article
- Published by Elsevier in Developmental Cell
- Vol. 53 (1) , 9-26.e4
Abstract
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Funding Information
- March of Dimes Foundation
- Eli and Edythe Broad Center of Regenerative Medicine and Stem Cell Research, University of California Los Angeles
- National Institute of Diabetes and Digestive and Kidney Diseases
This publication has 96 references indexed in Scilit:
- Phosphoglycerate dehydrogenase diverts glycolytic flux and contributes to oncogenesisNature Genetics, 2011
- Web-based inference of biological patterns, functions and pathways from metabolomic data using MetaboAnalystNature Protocols, 2011
- AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1Nature Cell Biology, 2011
- The S1P1-mTOR axis directs the reciprocal differentiation of TH1 and Treg cellsNature Immunology, 2010
- Structural insights into the YAP and TEAD complexGenes & Development, 2010
- GATA3 Is Selectively Expressed in the Trophectoderm of Peri-implantation Embryo and Directly Regulates Cdx2 Gene ExpressionJournal of Biological Chemistry, 2009
- Bone Marrow Progenitor Cells Induce Endothelial Adherens Junction Integrity by Sphingosine-1-Phosphate–Mediated Rac1 and Cdc42 SignalingCirculation Research, 2009
- The receptor S1P1 overrides regulatory T cell–mediated immune suppression through Akt-mTORNature Immunology, 2009
- Regulation of Translation Initiation in Eukaryotes: Mechanisms and Biological TargetsCell, 2009
- Inactivation of YAP oncoprotein by the Hippo pathway is involved in cell contact inhibition and tissue growth controlGenes & Development, 2007