Alteration of bile acid profile in patients with neuromyelitis optica spectrum disorders and multiple sclerosis
Authors:
- 23 August 2025
- journal article
- Published by Elsevier in Multiple Sclerosis and Related Disorders
- Vol. 103, 106703
Abstract
No abstract availableKeywords
Funding Information
- Chang Gung Memorial Hospital, Linkou
- Chang Gung Memorial Hospital (CMRPG3N0081-83)
This publication has 41 references indexed in Scilit:
- Update on the diagnosis and treatment of neuromyelitis optica spectrum disorders (NMOSD) – revised recommendations of the Neuromyelitis Optica Study Group (NEMOS). Part II: Attack therapy and long-term managementJournal of Neurology, 2023
- Hyocholic acid species improve glucose homeostasis through a distinct TGR5 and FXR signaling mechanismCell Metabolism, 2021
- Gut microbiome variation is associated to Multiple Sclerosis phenotypic subtypesAnnals of Clinical and Translational Neurology, 2020
- Neural sphingosine 1‐phosphate accumulation activates microglia and links impaired autophagy and inflammationGlia, 2019
- Glucocorticoid treatment alters systemic bile acid homeostasis by regulating the biosynthesis and transport of bile saltsDigestive and Liver Disease, 2016
- Biomarkers for neuromyelitis opticaClinica Chimica Acta, 2015
- Bile acids permeabilize the blood brain barrier after bile duct ligation in rats via Rac1-dependent mechanismsDigestive and Liver Disease, 2014
- Cerebrospinal fluid interleukin-6 and glial fibrillary acidic protein levels are increased during initial neuromyelitis optica attacksClinica Chimica Acta, 2013
- TGR5 Activation Inhibits Atherosclerosis by Reducing Macrophage Inflammation and Lipid LoadingCell Metabolism, 2011
- Conjugated Bile Acids Activate the Sphingosine–1–Phosphate Receptor 2 in Primary Rodent HepatocytesHepatology, 2011