The bone morphogenetic protein antagonist noggin protects white matter after perinatal hypoxia–ischemia
- 1 June 2011
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 42 (3) , 318-326
Abstract
No abstract availableKeywords
Funding Information
- NIH (KO8 NS053529)
- NIH NINDS (RO1 NS20013, NS20778)
This publication has 42 references indexed in Scilit:
- Hypoxia-Ischemia Induces an Endogenous Reparative Response by Local Neural Progenitors in the Postnatal Mouse TelencephalonDevelopmental Neuroscience, 2010
- Chordin-induced lineage plasticity of adult SVZ neuroblasts after demyelinationNature Neuroscience, 2010
- Noggin Protects Against Ischemic Brain Injury in RodentsStroke, 2010
- Noggin Expands Neural Stem Cells in the Adult HippocampusJournal of Neuroscience, 2008
- Adult Neurogenesis Requires Smad4-Mediated Bone Morphogenic Protein Signaling in Stem CellsThe Journal of Neuroscience, 2008
- BMPR1a Signaling Determines Numbers of Oligodendrocytes and Calbindin-Expressing Interneurons in the CortexJournal of Neuroscience, 2007
- Transgenic Mice Overexpressing BMP4 Develop a Fibrodysplasia Ossificans Progressiva (FOP)-Like PhenotypeThe American Journal of Pathology, 2004
- Bone Morphogenetic Protein Signaling Regulates Postnatal Hair Follicle Differentiation and CyclingThe American Journal of Pathology, 2004
- New oligodendrocytes are generated after neonatal hypoxic‐ischemic brain injury in rodentsGlia, 2004
- Effect of brain edema on infarct volume in a focal cerebral ischemia model in rats.Stroke, 1993