Delineation of the 1q24.3 microdeletion syndrome provides further evidence for the potential role of non-coding RNAs in regulating the skeletal phenotype
Authors:
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health
This publication has 42 references indexed in Scilit:
- MicroRNA-214 suppresses osteogenic differentiation of C2C12 myoblast cells by targeting OsterixBone, 2013
- MiR-3120 Is a Mirror MicroRNA That Targets Heat Shock Cognate Protein 70 and Auxilin Messenger RNAs and Regulates Clathrin Vesicle UncoatingJournal of Biological Chemistry, 2012
- Distinctive phenotype in 9 patients with deletion of chromosome 1q24‐q25American Journal of Medical Genetics Part A, 2011
- miR-199a*, a Bone Morphogenic Protein 2-responsive MicroRNA, Regulates Chondrogenesis via Direct Targeting to Smad1Journal of Biological Chemistry, 2009
- Allele-Specific Gene Expression Is Widespread Across the Genome and Biological ProcessesPLOS ONE, 2009
- MicroRNAs: Target Recognition and Regulatory FunctionsCell, 2009
- Twist-1 regulates the miR-199a/214 cluster during developmentNucleic Acids Research, 2008
- Dnm3os, a non‐coding RNA, is required for normal growth and skeletal development in miceDevelopmental Dynamics, 2008
- Postsynaptic Positioning of Endocytic Zones and AMPA Receptor Cycling by Physical Coupling of Dynamin-3 to HomerNeuron, 2007
- A conserved noncoding intronic transcript at the mouse Dnm3 locusGenomics, 2005