MiR-21-5p directly contributes to regulating eNOS expression in human artery endothelial cells under normoxia and hypoxia
- 1 December 2020
- journal article
- research article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 182, 114288
Abstract
No abstract availableKeywords
Funding Information
- Fondo Nacional de Desarrollo Científico y Tecnológico (1181341)
- Agencia de Innovación y Desarrollo de Andalucía
This publication has 62 references indexed in Scilit:
- Circulating MicroRNAs in Maternal Blood as Potential Biomarkers for Fetal Hypoxia In-UteroPLOS ONE, 2013
- In Vivo Monitoring of Angiogenesis Inhibition via Down-Regulation of Mir-21 in a VEGFR2-Luc Murine Breast Cancer Model Using Bioluminescent ImagingPLOS ONE, 2013
- Variable promoter methylation contributes to differential expression of key genes in human placenta-derived venous and arterial endothelial cellsBMC Genomics, 2013
- Reduced Cystathionine γ-Lyase and Increased miR-21 Expression Are Associated with Increased Vascular Resistance in Growth-Restricted PregnanciesThe American Journal of Pathology, 2013
- A novel source for miR-21 expression through the alternative polyadenylation of VMP1 gene transcriptsNucleic Acids Research, 2012
- Developmental Programming of Cardiovascular Dysfunction by Prenatal Hypoxia and Oxidative StressPLOS ONE, 2012
- Deep-sequencing of endothelial cells exposed to hypoxia reveals the complexity of known and novel microRNAsRNA, 2012
- Flow-Dependent Regulation of Krüppel-Like Factor 2 Is Mediated by MicroRNA-92aCirculation, 2011
- miR-16 and miR-21 Expression in the Placenta Is Associated with Fetal GrowthPLOS ONE, 2011
- A new mathematical model for relative quantification in real-time RT-PCRNucleic Acids Research, 2001