Th9 cells induce steroid-resistant bronchial hyperresponsiveness in mice
Authors:
Open Access
- 1 September 2017
- journal article
- Published by Elsevier in Allergology International
- Vol. 66, S35-S40
Abstract
No abstract availableKeywords
Funding Information
- Grant-in-Aid for JSPS KAKENHI (26860122)
- Grant-in-Aid for JSPS KAKENHI (15K07787)
This publication has 35 references indexed in Scilit:
- A Brief History of IL-9The Journal of Immunology, 2011
- IL-9 Promotes IL-13-Dependent Paneth Cell Hyperplasia and Up-Regulation of Innate Immunity Mediators in Intestinal MucosaThe Journal of Immunology, 2009
- IL-4 inhibits TGF-β-induced Foxp3+ T cells and, together with TGF-β, generates IL-9+ IL-10+ Foxp3− effector T cellsNature Immunology, 2008
- Selective suppression of Th2-mediated airway eosinophil infiltration by low-molecular weight CCR3 antagonistsInternational Immunology, 2007
- Oral corticosteroids decrease eosinophil and CC chemokine expression but increase neutrophil, IL-8, and IFN-γ–inducible protein 10 expression in asthmatic airway mucosaJournal of Allergy and Clinical Immunology, 2005
- Interleukin-9 Induces Goblet Cell Hyperplasia during Repair of Human Airway EpitheliaAmerican Journal of Respiratory Cell and Molecular Biology, 2003
- IMMUNOLOGIC BASIS OF ANTIGEN-INDUCED AIRWAY HYPERRESPONSIVENESSAnnual Review of Immunology, 1999
- Evidence that cyclosporin A and dexamethasone inhibit allergic airway eosinophilic inflammation via suppression of interleukin‐5 synthesis by T cellsBritish Journal of Pharmacology, 1998
- Absence of Glucocorticoid Receptor-β in MiceJournal of Biological Chemistry, 1997
- Predominant TH2-like Bronchoalveolar T-Lymphocyte Population in Atopic AsthmaNew England Journal of Medicine, 1992