The aortic valve microenvironment and its role in calcific aortic valve disease
- 1 May 2011
- journal article
- review article
- Published by Elsevier in Cardiovascular Pathology
- Vol. 20 (3) , 177-182
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Hypercholesterolemia Induces Side-Specific Phenotypic Changes and Peroxisome Proliferator–Activated Receptor-γ Pathway Activation in Swine Aortic Valve EndotheliumArteriosclerosis, Thrombosis, and Vascular Biology, 2010
- Endothelium-Dependent Regulation of the Mechanical Properties of Aortic Valve CuspsJACC, 2009
- Identification and Characterization of Aortic Valve Mesenchymal Progenitor Cells with Robust Osteogenic Calcification PotentialThe American Journal of Pathology, 2009
- Spatial Heterogeneity of Endothelial Phenotypes Correlates With Side-Specific Vulnerability to Calcification in Normal Porcine Aortic ValvesCirculation Research, 2005
- Smoothelin-positive cells in human and porcine semilunar valvesHistochemistry and Cell Biology, 2003
- Human Aortic Valve Calcification Is Associated With an Osteoblast PhenotypeCirculation, 2003
- Activated Interstitial Myofibroblasts Express Catabolic Enzymes and Mediate Matrix Remodeling in Myxomatous Heart ValvesCirculation, 2001
- Bone Formation and Inflammation in Cardiac ValvesCirculation, 2001
- Clinical Factors Associated With Calcific Aortic Valve DiseaseJACC, 1997
- Apolipoproteins B, (a), and E Accumulate in the Morphologically Early Lesion of ‘Degenerative’ Valvular Aortic StenosisArteriosclerosis, Thrombosis, and Vascular Biology, 1996