RAGE signaling mediates post-injury arterial neointima formation by suppression of liver kinase B1 and AMPK activity
- 1 June 2012
- journal article
- Published by Elsevier in Atherosclerosis
- Vol. 222 (2) , 417-425
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- AMPKα2 Deletion Exacerbates Neointima Formation by Upregulating Skp2 in Vascular Smooth Muscle CellsCirculation Research, 2011
- RAGE-Dependent Activation of the Oncoprotein Pim1 Plays a Critical Role in Systemic Vascular Remodeling ProcessesArteriosclerosis, Thrombosis, and Vascular Biology, 2011
- Signal Transducers and Activators of Transcription-3/Pim1 Axis Plays a Critical Role in the Pathogenesis of Human Pulmonary Arterial HypertensionCirculation, 2011
- Andrographolide Enhances Nuclear Factor-κB Subunit p65 Ser536 Dephosphorylation through Activation of Protein Phosphatase 2A in Vascular Smooth Muscle CellsJournal of Biological Chemistry, 2011
- Exogenous NAD Blocks Cardiac Hypertrophic Response via Activation of the SIRT3-LKB1-AMP-activated Kinase PathwayJournal of Biological Chemistry, 2010
- Chemokines in Vascular Dysfunction and RemodelingArteriosclerosis, Thrombosis, and Vascular Biology, 2008
- Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE–/– miceJournal of Clinical Investigation, 2008
- Suppression of oncogenic properties of c-Myc by LKB1-controlled epithelial organizationProceedings of the National Academy of Sciences, 2007
- Axl, A Receptor Tyrosine Kinase, Mediates Flow-Induced Vascular RemodelingCirculation Research, 2006
- AMP-activated protein kinase: Ancient energy gauge provides clues to modern understanding of metabolismCell Metabolism, 2005