Pathophysiology of the Blood–Spinal Cord Barrier in Traumatic Injury
Author:
- Hari Shanker SharmaHSHari Shanker Sharma
- 1 January 2004
- book chapter
- Published by Elsevier
- p. 437-518
Abstract
No abstract availableThis publication has 399 references indexed in Scilit:
- A new antioxidant compound H-290/51 attenuates nitric oxide synthase and heme oxygenase expression following hyperthermic brain injuryAmino Acids, 2000
- Neurotrophic factors influence upregulation of constitutive isoform of heme oxygenase and cellular stress response in the spinal cord following traumaAmino Acids, 2000
- Brain derived neurotrophic factor and insulin like growth factor-1 attenuate upregulation of nitric oxide synthase and cell injury following trauma to the spinal cordAmino Acids, 1998
- Astrocytes regulate GFAP mRNA levels by cyclic AMP and protein kinase C‐dependent mechanismsGlia, 1988
- Early membrane lipid changes in laminectomized and traumatized cat spinal cordNeurochemical Pathology, 1987
- The multimolecular cascade of spinal cord injuryNeurochemical Pathology, 1987
- Spinal cord extracellular microenvironmentNeurochemical Pathology, 1987
- The histamine-releasing effect of dynorphin and other peptides possessing Arg-Pro sequencesInflammation Research, 1985
- Effects ofp-chlorophenylalanine and a monoamine oxidase inhibitor on the 5-hydroxytryptamine in the spinal cord after transectionJournal of Neural Transmission, 1972
- Phospholipid precursors of prostaglandinsBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1968