PKCε Inhibits Neuronal Dendritic Spine Development through Dual Phosphorylation of Ephexin5
Open Access
- 1 November 2018
- journal article
- research article
- Published by Elsevier in Cell Reports
- Vol. 25 (9) , 2470-2483.e8
Abstract
No abstract availableKeywords
Funding Information
- National Institute of Mental Health (5R01MH102364-02)
- NIH (T32GM007445)
- National Science Foundation (1232825, 1746891)
This publication has 63 references indexed in Scilit:
- Phenotypical Analysis of Atypical PKCs In Vivo Function Display a Compensatory System at Mouse Embryonic Day 7.5PLOS ONE, 2013
- PKCα and PKCβ cooperate functionally in CD3-induced de novo IL-2 mRNA transcriptionImmunology Letters, 2013
- The Nogo Receptor Family Restricts Synapse Number in the Developing HippocampusNeuron, 2012
- A Tissue-Specific Atlas of Mouse Protein Phosphorylation and ExpressionCell, 2010
- EphB-Mediated Degradation of the RhoA GEF Ephexin5 Relieves a Developmental Brake on Excitatory Synapse FormationCell, 2010
- The substrates and binding partners of protein kinase CεBiochemical Journal, 2010
- Modulation of Rho Guanine Exchange Factor Lfc Activity by Protein Kinase A-Mediated PhosphorylationMolecular and Cellular Biology, 2009
- Cell Adhesion, the Backbone of the Synapse: "Vertebrate" and "Invertebrate" PerspectivesCold Spring Harbor Perspectives in Biology, 2009
- Regulation of AMPA receptor extrasynaptic insertion by 4.1N, phosphorylation and palmitoylationNature Neuroscience, 2009
- AMPA receptor and GEF-H1/Lfc complex regulates dendritic spine development through RhoA signaling cascadeProceedings of the National Academy of Sciences, 2009