2-iodohexadecanal induces autophagy during goiter involution
Authors:
- 11 February 2024
- journal article
- research article
- Published by Elsevier in Prostaglandins & Other Lipid Mediators
- Vol. 172, 106819
Abstract
No abstract availableKeywords
Funding Information
- CONICET
- FONCyT
This publication has 41 references indexed in Scilit:
- Complex Inhibitory Effects of Nitric Oxide on AutophagyMolecular Cell, 2011
- Inhibition of goiter growth and of cyclic AMP formation in rat thyroid by 2-iodohexadecanalMolecular and Cellular Endocrinology, 2010
- Signaling pathways involved in the antiproliferative effect of molecular iodine in normal and tumoral breast cells: evidence that 6-iodolactone mediates apoptotic effectsEndocrine-Related Cancer, 2008
- Iodine Deficiency Induces a Thyroid Stimulating Hormone-Independent Early Phase of Microvascular Reshaping in the ThyroidThe American Journal of Pathology, 2008
- Molecular Iodine Induces Caspase-independent Apoptosis in Human Breast Carcinoma Cells Involving the Mitochondria-mediated PathwayJournal of Biological Chemistry, 2006
- Apoptosis during goitre involution - the role of Bcl-2Journal of Endocrinology, 2000
- Presence and possible role of vascular endothelial growth factor in thyroid cell growth and functionJournal of Endocrinology, 1998
- Upregulation of the angiogenic factors PlGF, VEGF and their receptors (Flt-1, Flk-1/KDR) by TSH in cultured thyrocytes and in the thyroid gland of thiouracil-fed rats suggest a TSH-dependent paracrine mechanism for goiter hypervascularizationOncogene, 1997
- Biosynthesis and Metabolism of 2-Iodohexadecanal in Cultured Dog Thyroid CellsJournal of Biological Chemistry, 1996
- Control of thyroid cell proliferation and goitrogenesisTrends in Endocrinology & Metabolism, 1992