Clinical usefulness of measurement of fibroblast growth factor 23 (FGF23) in hypophosphatemic patients
Authors:
- Seiji FukumotoSFSeiji Fukumoto
- Toshimi MichigamiTMToshimi Michigami
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
- Ministry of Health, Labour and Welfare
This publication has 20 references indexed in Scilit:
- Effect of acute changes of serum phosphate on fibroblast growth factor (FGF)23 levels in humansJournal of Bone and Mineral Metabolism, 2007
- FGF23 Concentrations Vary With Disease Status in Autosomal Dominant Hypophosphatemic RicketsJournal of Bone and Mineral Research, 2007
- DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasisNature Genetics, 2006
- Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolismNature Genetics, 2006
- Regulation of C-Terminal and Intact FGF-23 by Dietary Phosphate in Men and WomenJournal of Bone and Mineral Research, 2006
- Comparison of two assays for fibroblast growth factor (FGF)-23Journal of Bone and Mineral Metabolism, 2005
- Circulating FGF-23 Is Regulated by 1α,25-Dihydroxyvitamin D3 and Phosphorus in VivoJournal of Biological Chemistry, 2005
- FGF-23 Is a Potent Regulator of Vitamin D Metabolism and Phosphate HomeostasisJournal of Bone and Mineral Research, 2004
- Fibroblast Growth Factor 23 in Oncogenic Osteomalacia and X-Linked HypophosphatemiaNew England Journal of Medicine, 2003
- NOMOGRAM FOR DERIVATION OF RENAL THRESHOLD PHOSPHATE CONCENTRATIONThe Lancet, 1975