Endocannabinoids at the spinal level regulate, but do not mediate, nonopioid stress-induced analgesia

This publication has 59 references indexed in Scilit:

• Lipids as regulators of the activity of transient receptor potential type V1 (TRPV1) channels
  Life Sciences, 2005
• Fatty acid amide hydrolase: an emerging therapeutic target in the endocannabinoid system
  Current Opinion in Chemical Biology, 2003
• Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase
  Proceedings of the National Academy of Sciences, 2001
• The Vanilloid Receptor: A Molecular Gateway to the Pain Pathway
  Annual Review of Neuroscience, 2001
• The cannabinoid CB₁ receptor antagonist, SR141716A, selectively facilitates nociceptive responses of dorsal horn neurones in the rat
  British Journal of Pharmacology, 1999
• Arachidonoylserotonin and Other Novel Inhibitors of Fatty Acid Amide Hydrolase
  Biochemical and Biophysical Research Communications, 1998
• The capsaicin receptor: a heat-activated ion channel in the pain pathway
  Nature, 1997
• Biosynthesis, Uptake, and Degradation of Anandamide and Palmitoylethanolamide in Leukocytes
  Journal of Biological Chemistry, 1997
• Molecular characterization of an enzyme that degrades neuromodulatory fatty-acid amides
  Nature, 1996
• The Many Possible Roles of Opioids and Related Peptides in Stress‐Induced Analgesia^a
  Annals of the New York Academy of Sciences, 1986