CD34+ PI16+ fibroblast progenitors aggravate neointimal lesions of allograft arteries via CCL11/CCR3-PI3K/AKT pathway
Authors:
Open Access
- 20 January 2025
- journal article
- research article
- Published by Ivyspring International Publisher in Theranostics
- Vol. 15 (6) , 2523-2543
Abstract
Rationale: Transplant-accelerated arteriosclerosis is a common complication that limits the long-term survival of organ transplant recipients. While previous studies have indicated the involvement of CD34+ stem/progenitor cells (SPCs) in this process, their heterogeneity and potential adverse effects remains incompletely understood.Keywords
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