Enhanced nitric oxide production during lead (Pb2+) exposure recovers protein expression but not presynaptic localization of synaptic proteins in developing hippocampal neurons

This publication has 48 references indexed in Scilit:

• Lead exposure during synaptogenesis alters NMDA receptor targeting via NMDA receptor inhibition
  NeuroToxicology, 2011
• Lead exposure alters cyclic-AMP response element binding protein phosphorylation and binding activity in the developing rat brain
  Developmental Brain Research, 2003
• Opposing roles of synaptic and extrasynaptic NMDA receptors in neuronal calcium signalling and BDNF gene regulation
  Current Opinion in Neurobiology, 2003
• Developmental Pb2+ exposure alters NMDAR subtypes and reduces CREB phosphorylation in the rat brain
  Developmental Brain Research, 2002
• Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways
  Nature Neuroscience, 2002
• Ca2+-dependent Formation of a Dynamin-Synaptophysin Complex
  Journal of Biological Chemistry, 2002
• Effect of developmental lead exposure on the expression of specific NMDA receptor subunit mRNAs in the hippocampus of neonatal rats by digoxigenin-labeled in situ hybridization histochemistry
  Neurotoxicology and Teratology, 2002
• SNARE Function Analyzed in Synaptobrevin/VAMP Knockout Mice
  Science, 2001
• Synaptic secretion of BDNF after high‐frequency stimulation of glutamatergic synapses
  The EMBO Journal, 2001
• N-methyl-d-aspartate receptor subunit changes are associated with lead-induced deficits of long-term potentiation and spatial learning
  Neuroscience, 2000