Induction of Survivin Expression by Taxol (Paclitaxel) Is an Early Event, Which Is Independent of Taxol-mediated G2/M Arrest
Authors:
- Ralph J. BernackiRBRalph J. Bernacki
- Michael G. BrattainMBMichael G. Brattain
- Fengzhi LiFengzhi Li
Open Access
- 1 April 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (15) , 15196-15203
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Survivin study: What is the next wave?Journal of Cellular Physiology, 2003
- Regulation of apoptosis at cell division by p34 cdc2 phosphorylation of survivinProceedings of the National Academy of Sciences, 2000
- Down-regulation of Survivin by Antisense Oligonucleotides Increases Apoptosis, Inhibits Cytokinesis and Anchorage-Independent GrowthNeoplasia, 2000
- Pleiotropic cell-division defects and apoptosis induced by interference with survivin functionNature Cell Biology, 1999
- Control of apoptosis and mitotic spindle checkpoint by survivinNature, 1998
- Induction of Apoptosis and Inhibition of Cell Proliferation bysurvivin Gene TargetingJournal of Biological Chemistry, 1998
- PaclitaxelDrugs & Aging, 1998
- The TaxoidsDrugs, 1998
- Paclitaxel (Taxol)New England Journal of Medicine, 1995
- Cytotoxic studies of paclitaxel (Taxol®) in human tumour cell linesBritish Journal of Cancer, 1993