SOCS2 overexpression alleviates diabetic nephropathy in rats by inhibiting the TLR4/NF-κB pathway
Authors:
Open Access
- 24 August 2017
- journal article
- Published by Impact Journals, LLC in Oncotarget
- Vol. 8 (53) , 91185-91198
Abstract
SOCS2 overexpression alleviates diabetic nephropathy in rats by inhibiting the TLR4/NF-B pathway SOCS2 overexpression alleviates diabetic nephropathy in rats by inhibiting the TLR4/NF-κB pathwayKeywords
This publication has 29 references indexed in Scilit:
- Inflammation in Diabetic NephropathyMediators of Inflammation, 2012
- Toll-Like Receptor 4 Promotes Tubular Inflammation in Diabetic NephropathyJournal of the American Society of Nephrology, 2012
- STAT5A-Mediated SOCS2 Expression Regulates Jak2 and STAT3 Activity Following c-Src Inhibition in Head and Neck Squamous CarcinomaClinical Cancer Research, 2012
- Toll-like receptor 4 regulates early endothelial activation during ischemic acute kidney injuryKidney International, 2011
- TLR4 Promotes Fibrosis but Attenuates Tubular Damage in Progressive Renal InjuryJournal of the American Society of Nephrology, 2010
- Suppressors of Cytokine Signaling Abrogate Diabetic NephropathyJournal of the American Society of Nephrology, 2010
- Improved Elucidation of Biological Processes Linked to Diabetic Nephropathy by Single Probe-Based Microarray Data AnalysisPLOS ONE, 2008
- Human intrahepatic biliary epithelial cells function in innate immunity by producing IL‐6 and IL‐8 via the TLR4‐NF‐κB and ‐MAPK signaling pathwaysLiver International, 2006
- The Crystal Structure of the IκBα/NF-κB Complex Reveals Mechanisms of NF-κB InactivationCell, 1998
- Rearrangements of the Cytoskeleton and Cell Contacts Induce Process Formation during Differentiation of Conditionally Immortalized Mouse Podocyte Cell LinesExperimental Cell Research, 1997