Pratensein ameliorates β-amyloid-induced cognitive impairment in rats via reducing oxidative damage and restoring synapse and BDNF levels
Authors:
- 1 April 2015
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 592, 48-53
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (81260674, 81260505)
- Guangxi Natural Science Foundation (2013GXNSFAA019150, 2013GXNSFAA019146)
This publication has 24 references indexed in Scilit:
- FTY720 (Fingolimod) Attenuates Beta-amyloid Peptide (Aβ42)-Induced Impairment of Spatial Learning and Memory in RatsJournal of Molecular Neuroscience, 2013
- Antioxidant Profile of Trifolium pratense L.Molecules, 2012
- Indomethacin-loaded lipid-core nanocapsules reduce the damage triggered by Aβ1-42 in Alzheimer’s disease modelsInternational Journal of Nanomedicine, 2012
- Inflammation in the early stages of neurodegenerative pathologyJournal of Neuroimmunology, 2011
- Enhancement of LTP in Aged Rats is Dependent on Endogenous BDNFNeuropsychopharmacology, 2011
- Tetrandrine attenuates spatial memory impairment and hippocampal neuroinflammation via inhibiting NF-κB activation in a rat model of Alzheimer's disease induced by amyloid-β(1–42)Brain Research, 2011
- Protective effect of isoflavones from Trifolium pratense on dopaminergic neuronsNeuroscience Research, 2008
- Alzheimer's diseaseThe Lancet, 2006
- The NF-κB/Rel family of proteins mediates Aβ-induced neurotoxicity and glial activationMolecular Brain Research, 1998
- Nuclear translocation of NF-κB in cholinergic neurons of patients with Alzheimerʼs diseaseNeuroReport, 1997