HIV-1 Tat upregulates expression of histone deacetylase-2 (HDAC2) in human neurons: Implication for HIV-associated neurocognitive disorder (HAND)
- 1 May 2011
- journal article
- Published by Elsevier in Neurochemistry International
- Vol. 58 (6) , 656-664
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- Histone deacetylase inhibitors and HIV latencyCurrent Opinion in HIV and AIDS, 2011
- HIV-associated neurocognitive disorders: is there a hidden epidemic?AIDS, 2010
- Chromatin dynamics associated with HIV-1 Tat-activated transcriptionBiochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms, 2010
- Multiple roles of HDAC inhibition in neurodegenerative conditionsTrends in Neurosciences, 2009
- Cognitive Neuropsychology of HIV-Associated Neurocognitive DisordersNeuropsychology Review, 2009
- A Limited Group of Class I Histone Deacetylases Acts To Repress Human Immunodeficiency Virus Type 1 ExpressionJournal of Virology, 2009
- HDAC2 negatively regulates memory formation and synaptic plasticityNature, 2009
- The Challenge of Finding a Cure for HIV InfectionScience, 2009
- Expression of Latent HIV Induced by the Potent HDAC Inhibitor Suberoylanilide Hydroxamic AcidAIDS Research and Human Retroviruses, 2009
- HIV-1 gp120 Compromises Blood–Brain Barrier Integrity and Enhance Monocyte Migration across Blood–Brain Barrier: Implication for Viral NeuropathogenesisJournal of Cerebral Blood Flow & Metabolism, 2007