Indomethacin Sensitizes TRAIL-Resistant Melanoma Cells to TRAIL-Induced Apoptosis through ROS-Mediated Upregulation of Death Receptor 5 and Downregulation of Survivin

This publication has 55 references indexed in Scilit:

• Sensitization of melanoma cells for TRAIL-induced apoptosis by BMS-345541 correlates with altered phosphorylation and activation of Bax
  Cell Death & Disease, 2013
• New therapeutical strategies in the treatment of metastatic disease
  Dermatologic Therapy, 2012
• General Sensitization of Melanoma Cells for TRAIL-Induced Apoptosis by the Potassium Channel Inhibitor TRAM-34 Depends on Release of SMAC
  PLOS ONE, 2012
• In the Huh7 Hepatoma Cells Diclofenac and Indomethacin Activate Differently the Unfolded Protein Response and Induce ER Stress Apoptosis
  The Open Biochemistry Journal, 2011
• Inhibition of androgen receptor activity by histone deacetylase 4 through receptor SUMOylation
  Oncogene, 2011
• ROS and CHOP Are Critical for Dibenzylideneacetone to Sensitize Tumor Cells to TRAIL through Induction of Death Receptors and Downregulation of Cell Survival Proteins
  Cancer Research, 2011
• Garcinol Potentiates TRAIL-Induced Apoptosis through Modulation of Death Receptors and Antiapoptotic Proteins
  Molecular Cancer Therapeutics, 2010
• Death-receptor O-glycosylation controls tumor-cell sensitivity to the proapoptotic ligand Apo2L/TRAIL
  Nature Medicine, 2007
• Cutaneous Malignant Melanoma
  Mayo Clinic Proceedings, 2006
• Endoplasmic reticulum stress response is involved in nonsteroidal anti-inflammatory drug-induced apoptosis
  Cell Death & Differentiation, 2004