Diet-induced death: microbiome-mediated lipid stress reduces intestinal ILC3s
- 25 March 2026
- journal article
- Published by Elsevier in Trends in Immunology
- Vol. 47 (4) , 252-254
Abstract
No abstract availableKeywords
Funding Information
- Burroughs Wellcome Fund
- National Institutes of Health (R01AI123368)
- National Institutes of Health (R37AI174468)
- National Institutes of Health (R01AI162936)
- National Institutes of Health (R01CA274534)
- National Institutes of Health (R01AI143842)
- National Institutes of Health (R01AI145989)
- National Institutes of Health (U01AI095608)
- Cancer Research Institute
This publication has 10 references indexed in Scilit:
- High-fat diet causes rapid loss of intestinal group 3 innate lymphoid cells through microbiota-driven inflammation and mitochondrial stressImmunity, 2026
- Interleukin-23 biology linking mucosal immunity to autoimmune diseases and cancerTrends in Immunology, 2026
- Acute exposure to high-fat diet impairs ILC3 functions and gut homeostasisImmunity, 2025
- RIPK3 and caspase-8 interpret cytokine signals to regulate ILC3 survival in the gutMucosal Immunology, 2024
- Group 3 innate lymphoid cells in intestinal health and diseaseNature Reviews Gastroenterology & Hepatology, 2024
- Intestinal IL-22RA1 signaling regulates intrinsic and systemic lipid and glucose metabolism to alleviate obesity-associated disordersNature Communications, 2024
- Microbiota imbalance induced by dietary sugar disrupts immune-mediated protection from metabolic syndromeCell, 2022
- Innate and adaptive lymphocytes sequentially shape the gut microbiota and lipid metabolismNature, 2018
- Interleukin-22 alleviates metabolic disorders and restores mucosal immunity in diabetesNature, 2014
- Interaction Between Obesity and the Gut Microbiota: Relevance in NutritionAnnual Review of Nutrition, 2011