Hypoxia-inducible factor-1α regulates microglial functions affecting neuronal survival in the acute phase of ischemic stroke in mice
Authors:
Open Access
- 1 December 2017
- journal article
- research article
- Published by Impact Journals, LLC in Oncotarget
- Vol. 8 (67) , 111508-111521
Abstract
Hypoxia-inducible factor-1 regulates microglial functions affecting neuronal survival in the acute phase of ischemic stroke in mice Hypoxia-inducible factor-1α regulates microglial functions affecting neuronal survival in the acute phase of ischemic stroke in miceKeywords
This publication has 32 references indexed in Scilit:
- Cell Surface CD36 Protein in Monocyte/Macrophage Contributes to Phagocytosis during the Resolution Phase of Ischemic Stroke in MiceJournal of Biological Chemistry, 2016
- Macrophage depletion reduced brain injury following middle cerebral artery occlusion in miceJournal of Neuroinflammation, 2016
- The HIF-1/glial TIM-3 axis controls inflammation-associated brain damage under hypoxiaNature Communications, 2015
- PET Imaging of Stroke-Induced Neuroinflammation in Mice Using [18F]PBR06Molecular Imaging and Biology, 2013
- Information processing during phagocytosisNature Reviews Immunology, 2012
- Biomarkers of Therapeutic Response in the IL-23 Pathway in Inflammatory Bowel DiseaseClinical and Translational Gastroenterology, 2012
- Physiology of MicrogliaPhysiological Reviews, 2011
- Hypoxia-induced iNOS expression in microglia is regulated by the PI3-kinase/Akt/mTOR signaling pathway and activation of hypoxia inducible factor-1αBiochemical Pharmacology, 2006
- The Class B Scavenger Receptor CD36 Mediates Free Radical Production and Tissue Injury in Cerebral IschemiaJournal of Neuroscience, 2005
- HIF-1α Is Essential for Myeloid Cell-Mediated InflammationCell, 2003