The role of Branched Chain Amino Acids in the treatment of hepatic Encephalopathy
- 1 December 2018
- journal article
- review article
- Published by Elsevier in Journal of Clinical and Experimental Hepatology
- Vol. 8 (4) , 448-451
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Branched-chain amino acids increase arterial blood ammonia in spite of enhanced intrinsic muscle ammonia metabolism in patients with cirrhosis and healthy subjectsAmerican Journal of Physiology-Gastrointestinal and Liver Physiology, 2011
- Acute hyperammonemia activates branched-chain amino acid catabolism and decreases their extracellular concentrations: different sensitivity of red and white muscleAmino Acids, 2010
- Variation in blood ammonia concentration with site of measurement and evidence of brain and muscle uptake in patients with acute liver failureLiver International, 2008
- Direct molecular and spectroscopic evidence for increased ammonia removal capacity of skeletal muscle in acute liver failureJournal of Hepatology, 2006
- Blood–brain barrier permeability to ammonia in liver failure: a critical reappraisalNeurochemistry International, 2004
- Hyperammonemia-induced depletion of glutamate and branched-chain amino acids in muscle and plasmaJournal of Hepatology, 1996
- Comparison of Dietary Protein with an Oral, Branched Chain-Enriched Amino Acid Supplement in Chronic Portal-Systemic Encephalopathy: A Randomized Controlled TrialHepatology, 1984
- Significance of intra- and extrahepatic portasystemic shunting in survival of cirrhotic patientsDigestive Diseases and Sciences, 1981
- Synthesis of urea after stimulation with amino acids: relation to liver function.Gut, 1980
- Die Eck'sche Fistel zwischen der unteren Hohlvene und der Pfortader und ihre Folgen für den OrganismusNaunyn-Schmiedeberg's Archives of Pharmacology, 1893