SGK1 inhibition induces fetal hemoglobin expression and delays polymerization in sickle erythroid cells
Open Access
- 24 May 2023
- journal article
- letter
- Published by American Society of Hematology in Blood Advances
- Vol. 7 (11) , 2317-2323
Abstract
No abstract availableThis publication has 27 references indexed in Scilit:
- Dimethyl fumarate increases fetal hemoglobin, provides heme detoxification, and corrects anemia in sickle cell diseaseJCI Insight, 2017
- Phosphorylation and acetylation modifications of FOXO3a: Independently or synergistically?Oncology Letters, 2017
- Crizanlizumab for the Prevention of Pain Crises in Sickle Cell DiseaseNew England Journal of Medicine, 2017
- Hemopexin therapy reverts heme-induced proinflammatory phenotypic switching of macrophages in a mouse model of sickle cell diseaseBlood, 2016
- Serum and Glucocorticoid–Regulated Kinase 1 Regulates Neutrophil Clearance during Inflammation ResolutionThe Journal of Immunology, 2014
- Evaluation of Th17 related cytokines associated with clinical and laboratorial parameters in sickle cell anemia patients with leg ulcersCytokine, 2014
- Second AKT: The rise of SGK in cancer signallingGrowth Factors, 2010
- SGK KINASES AND THEIR ROLE IN EPITHELIAL TRANSPORTAnnual Review of Physiology, 2006
- Protein Kinase SGK Mediates Survival Signals by Phosphorylating the Forkhead Transcription Factor FKHRL1 (FOXO3a)Molecular and Cellular Biology, 2001
- Role of gamma 87 Gln in the inhibition of hemoglobin S polymerization by hemoglobin FJournal of Biological Chemistry, 1994