Leptin receptor-induced STAT3-independent signaling pathways are protective against atherosclerosis in a murine model of obesity and hyperlipidemia

This publication has 28 references indexed in Scilit:

• Monocyte Chemoattractant Protein-1 Deficiency Protects Against Visceral Fat-Induced Atherosclerosis
  Arteriosclerosis, Thrombosis, and Vascular Biology, 2010
• Enhanced Orexin Receptor-2 Signaling Prevents Diet-Induced Obesity and Improves Leptin Sensitivity
  Cell Metabolism, 2009
• Leptin-induced endothelial dysfunction in obesity
  American Journal of Physiology-Heart and Circulatory Physiology, 2008
• Leptin activates AMP-activated protein kinase in hepatic cells via a JAK2-dependent pathway
  Biochemical and Biophysical Research Communications, 2006
• Increased hypercholesterolemia and atherosclerosis in mice lacking both ApoE and leptin receptor
  Atherosclerosis, 2005
• Recombinant Leptin Promotes Atherosclerosis and Thrombosis in Apolipoprotein E–Deficient Mice
  Arteriosclerosis, Thrombosis, and Vascular Biology, 2005
• Combined Adipocyte-Macrophage Fatty Acid–Binding Protein Deficiency Improves Metabolism, Atherosclerosis, and Survival in Apolipoprotein E–Deficient Mice
  Circulation, 2004
• STAT3 signalling is required for leptin regulation of energy balance but not reproduction
  Nature, 2003
• Severe Hypercholesterolemia, Hypertriglyceridemia, and Atherosclerosis in Mice Lacking Both Leptin and the Low Density Lipoprotein Receptor
  Journal of Biological Chemistry, 2001
• Obesity as an independent risk factor for cardiovascular disease: a 26-year follow-up of participants in the Framingham Heart Study.
  Circulation, 1983