Glioma sensitive or chemoresistant to temozolomide differentially modulate macrophage protumor activities
- 1 November 2017
- journal article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - General Subjects
- Vol. 1861 (11) , 2652-2662
Abstract
No abstract availableKeywords
Funding Information
- Conselho Nacional de Desenvolvimento Científico e Tecnológico (310846/2014-5, 482055/2013-8)
- Fundação de Amparo à Pesquisa do Estado do Rio Grande do Sul (16/2551-0000265-7, 16/2551-0000473-0)
- Coordenação de Aperfeiçoamento de Pessoal de Nível Superior
This publication has 34 references indexed in Scilit:
- Production of Adenosine by Ectonucleotidases: A Key Factor in Tumor ImmunoescapeJournal of Biomedicine and Biotechnology, 2012
- Microglia/macrophages promote glioma progressionGlia, 2010
- Acquisition of Temozolomide Chemoresistance in Gliomas Leads to Remodeling of Mitochondrial Electron Transport ChainJournal of Biological Chemistry, 2010
- Activation of AMP-activated Protein Kinase by Temozolomide Contributes to Apoptosis in Glioblastoma Cells via p53 Activation and mTORC1 InhibitionJournal of Biological Chemistry, 2010
- Lung Cancer Cell Lines as Tools for Biomedical Discovery and ResearchJNCI: Journal of the National Cancer Institute, 2010
- Genetic alterations and signaling pathways in the evolution of gliomasCancer Science, 2009
- Selective NTPDase2 expression modulates in vivo rat glioma growthCancer Science, 2009
- Classical and alternative activation of mononuclear phagocytes: Picking the best of both worlds for tumor promotionImmunobiology, 2006
- Nitric oxide. A macrophage product responsible for cytostasis and respiratory inhibition in tumor target cells.Journal of Experimental Medicine, 1989
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976