Expression of Stress-Response and Cell Proliferation Genes in Renal Cell Carcinoma Induced by Oxidative Stress
Authors:
- Shohei KondoSKShohei Kondo
- Yoko IwasaYIYoko Iwasa
- Hiroshi HiaiHHHiroshi Hiai
- Shinya ToyokuniSTShinya Toyokuni
- 1 June 2000
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 156 (6) , 2149-2157
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 65 references indexed in Scilit:
- The chemokine receptor CXCR4 is essential for vascularization of the gastrointestinal tractNature, 1998
- Establishment and Characterization of Three Androgen‐independent, Metastatic Carcinoma Cell Lines from 3,2′‐Dimethyl‐4‐aminobiphenyl‐induced Prostatic Tumors in F344 RatsJapanese Journal of Cancer Research, 1996
- Genomes with distinct function compositionFEBS Letters, 1996
- The Y-box Motif Mediates Redox-dependent Transcriptional Activation in Mouse CellsJournal of Biological Chemistry, 1995
- Low Incidence of Point Mutations in H‐, K‐ and N‐ras Oncogenes and p53 Tumor Suppressor Gene in Renal Cell Carcinoma and Peritoneal Mesothelioma of Wistar Rats Induced by Ferric NitrilotriacetateJapanese Journal of Cancer Research, 1995
- Renal carcinogenesis in the Eker ratJournal of Cancer Research and Clinical Oncology, 1995
- Over‐expression of the S13 ribosomal protein in actively growing cellsInternational Journal of Cancer, 1993
- Expression and purification of a trefoil peptide motif in a β‐galactosidase fusion protein and its use to search for trefoil‐binding sitesEuropean Journal of Biochemistry, 1993
- Protein Tyrosine Phosphatases: A Diverse Family of Intracellular and Transmembrane EnzymesScience, 1991
- High constitutive transcription of HSP86 gene in murine embryonal carcinoma cellsDifferentiation, 1989