Hereditary angioedema attack: what happens to vasoactive mediators?
Open Access
- 1 January 2020
- journal article
- research article
- Published by Elsevier in International Immunopharmacology
- Vol. 78, 106079
Abstract
No abstract availableKeywords
Funding Information
- Regione Campania
This publication has 25 references indexed in Scilit:
- Innate effector cells in angiogenesis and lymphangiogenesisCurrent Opinion in Immunology, 2018
- Angioedema Due to Bradykinin DysregulationThe Journal of Allergy and Clinical Immunology: In Practice, 2018
- The international WAO/EAACI guideline for the management of hereditary angioedema—The 2017 revision and updateAllergy, 2018
- Endothelial cell activation during edematous attacks of hereditary angioedema types I and IIJournal of Allergy and Clinical Immunology, 2014
- High‐molecular‐weight kininogen cleavage correlates with disease states in the bradykinin‐mediated angioedema due to hereditary C1‐inhibitor deficiencyClinical & Experimental Allergy, 2014
- Immune Cells as a Source and Target of Angiogenic and Lymphangiogenic Factors2013
- The plasma bradykinin-forming pathways and its interrelationships with complementMolecular Immunology, 2010
- Molecular mechanisms of endothelial hyperpermeability: implications in inflammationExpert Reviews in Molecular Medicine, 2009
- Isolation of Angiopoietin-1, a Ligand for the TIE2 Receptor, by Secretion-Trap Expression CloningCell, 1996
- Tumor Cells Secrete a Vascular Permeability Factor That Promotes Accumulation of Ascites FluidScience, 1983