Reduction of p53 gene dosage does not increase initiation or promotion but enhances malignant progression of chemically induced skin tumors
Authors:
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumoursNature, 1992
- p53 functions as a cell cycle control protein in osteosarcomas.Molecular and Cellular Biology, 1990
- Suppression of Human Colorectal Carcinoma Cell Growth by Wild-Type p53Science, 1990
- A genetic model for colorectal tumorigenesisCell, 1990
- Genetic changes in skin tumor progression: Correlation between presence of a mutant ras gene and loss of heterozygosity on mouse chromosome 7Cell, 1990
- Chromosome 17 Deletions and p53 Gene Mutations in Colorectal CarcinomasScience, 1989
- Oncogene Activation in Chemical Carcinogenesis1988
- ras GENESAnnual Review of Biochemistry, 1987
- Microinjection of the ras oncogene protein into PC12 cells induces morphological differentiationCell, 1985
- Activation of the mouse cellular Harvey-ras gene in chemically induced benign skin papillomasNature, 1984