Peroxiredoxin-1 regulates lipid peroxidation in corneal endothelial cells
Open Access
- 1 February 2020
- journal article
- research article
- Published by Elsevier in Redox Biology
- Vol. 30, 101417
Abstract
No abstract availableKeywords
Funding Information
- Singapore National Medical Research Council (NMRC), Clinician Scientist Award (NMRC/CSA-INV/0004/2015)
This publication has 32 references indexed in Scilit:
- Association of the Gutta-Induced Microenvironment With Corneal Endothelial Cell Behavior and Demise in Fuchs Endothelial Corneal DystrophyJAMA Ophthalmology, 2018
- Ferroptosis: A Regulated Cell Death Nexus Linking Metabolism, Redox Biology, and DiseaseCell, 2017
- Nrf2-Keap1 pathway promotes cell proliferation and diminishes ferroptosisOncogenesis, 2017
- Peroxiredoxin 1 Protects Telomeres from Oxidative Damage and Preserves Telomeric DNA for Extension by TelomeraseCell Reports, 2016
- Fuchs endothelial corneal dystrophy: current perspectivesClinical Ophthalmology, 2016
- Propagation of Human Corneal Endothelial Cells: A Novel Dual Media ApproachCell Transplantation, 2015
- Transethnic Replication of Association of CTG18.1 Repeat Expansion ofTCF4Gene With Fuchs' Corneal Dystrophy in Chinese Implies Common Causal VariantInvestigative Ophthalmology & Visual Science, 2014
- Decreased DJ-1 Leads to Impaired Nrf2-Regulated Antioxidant Defense and Increased UV-A–Induced Apoptosis in Corneal Endothelial CellsInvestigative Ophthalmology & Visual Science, 2014
- The Nrf2 regulatory network provides an interface between redox and intermediary metabolismTrends in Biochemical Sciences, 2014
- Inactivation of Peroxiredoxin I by Phosphorylation Allows Localized H2O2 Accumulation for Cell SignalingCell, 2010