Inhibition of STAT5: A therapeutic option in BCR-ABL1-driven leukemia
Open Access
- 9 October 2014
- journal article
- review article
- Published by Impact Journals, LLC in Oncotarget
- Vol. 5 (20) , 9564-9576
Abstract
Inhibition of STAT5: A therapeutic option in BCR-ABL1-driven leukemia Inhibition of STAT5: A therapeutic option in BCR-ABL1-driven leukemiaKeywords
This publication has 102 references indexed in Scilit:
- CDK8-Mediated STAT1-S727 Phosphorylation Restrains NK Cell Cytotoxicity and Tumor SurveillanceCell Reports, 2013
- CDK8 Kinase Phosphorylates Transcription Factor STAT1 to Selectively Regulate the Interferon ResponseImmunity, 2013
- Characterization of EHop-016, Novel Small Molecule Inhibitor of Rac GTPaseJournal of Biological Chemistry, 2012
- Phase I trial of the combination of flavopiridol and imatinib mesylate in patients with Bcr-Abl+ hematological malignanciesCancer Chemotherapy and Pharmacology, 2012
- Diverging fates of cells of origin in acute and chronic leukaemiaEMBO Molecular Medicine, 2012
- Ran‐dependent nuclear export mediators: a structural perspectiveThe EMBO Journal, 2011
- Stat5 is indispensable for the maintenance of bcr/abl‐positive leukaemiaEMBO Molecular Medicine, 2010
- The Btk tyrosine kinase is a major target of the Bcr-Abl inhibitor dasatinibProceedings of the National Academy of Sciences, 2007
- Rac1 and a GTPase-activating protein, MgcRacGAP, are required for nuclear translocation of STAT transcription factorsJournal of Cell Biology, 2006
- A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia veraNature, 2005