Cell proliferation and survival mechanisms underlying the abnormal persistence of follicular cysts in bovines with cystic ovarian disease induced by ACTH
Authors:
- Matías L. StangaferroMSMatías L. Stangaferro
- Martín M. PalomarMPMartín M. Palomar
- Natalia S. AlfaroNANatalia S. Alfaro
- Florencia ReyFRFlorencia Rey
- Eduardo J. GimenoEGEduardo J. Gimeno
- Hugo H. OrtegaHOHugo H. Ortega
- 1 October 2010
- journal article
- Published by Elsevier in Animal Reproduction Science
- Vol. 122 (1-2) , 98-110
Abstract
No abstract availableKeywords
This publication has 55 references indexed in Scilit:
- An imbalance between apoptosis and proliferation contributes to follicular persistence in polycystic ovaries in ratsReproductive Biology and Endocrinology, 2009
- Insulin-like growth factor I in sera, ovarian follicles and follicular fluid of cows with spontaneous or induced cystic ovarian diseaseResearch in Veterinary Science, 2008
- Quantification of diverse subcellular immunohistochemical markers with clinicobiological relevancies: validation of a new computer‐assisted image analysis procedureJournal of Anatomy, 2008
- Granulosa Cell Survival and Proliferation Are Altered in Polycystic Ovary SyndromeThe Journal of Clinical Endocrinology & Metabolism, 2008
- Estrogen Receptors α and β and Progesterone Receptors in Normal Bovine Ovarian Follicles and Cystic Ovarian DiseaseVeterinary Pathology, 2007
- Intraovarian Localization of Growth Factors in Induced Cystic Ovaries in RatsAnatomia, Histologia, Embryologia, 2007
- Estrous cycle dependent changes in expression and distribution of Fas, Fas ligand, Bcl-2, Bax, and pro- and active caspase-3 in the rat ovaryJournal of Endocrinology, 2006
- Expression of Cytoskeletal Proteins in the Follicular Wall of Induced Ovarian CystsCells Tissues Organs, 2004
- Dominant Bovine Ovarian Follicular Cysts Express Increased Levels of Messenger RNAs for Luteinizing Hormone Receptor and 3β-Hydroxysteroid Dehydrogenase Δ4,Δ5 Isomerase Compared to Normal Dominant FolliclesBiology of Reproduction, 2001
- Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation.Journal of Cell Biology, 1992