Depletion of Werner helicase results in mitotic hyperrecombination and pleiotropic homologous and nonhomologous recombination phenotypes

This publication has 75 references indexed in Scilit:

• Sgs1 Regulates Gene Conversion Tract Lengths and Crossovers Independently of Its Helicase Activity
  Molecular and Cellular Biology, 2006
• Loss of Werner syndrome protein function promotes aberrant mitotic recombination
  Genes & Development, 2001
• Werner syndrome cells are sensitive to DNA cross‐linking drugs
  The FASEB Journal, 2001
• Role of ERCC1 in removal of long non‐homologous tails during targeted homologous recombination
  The EMBO Journal, 2000
• Role of the nucleotide excision repair gene ERCC1 in formation of recombination-dependent rearrangements in mammalian cells
  Nucleic Acids Research, 2000
• Functional Interaction between Ku and the Werner Syndrome Protein in DNA End Processing
  Journal of Biological Chemistry, 2000
• Homologous recombination is responsible for cell death in the absence of the Sgs1 and Srs2 helicases
  Nature Genetics, 2000
• Partial suppression of the fission yeast rqh1− phenotype by expression of a bacterial Holliday junction resolvase
  The EMBO Journal, 2000
• Ku complex interacts with and stimulates the Werner protein
  Genes & Development, 2000
• Determining Mutation Rates in Bacterial Populations
  Methods, 2000