Genetic Deletion of the Tumor Necrosis Factor Receptor p60 or p80 Abrogates Ligand-mediated Activation of Nuclear Factor-κB and of Mitogen-activated Protein Kinases in Macrophages
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Open Access
- 1 August 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (34) , 31906-31912
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Tumour necrosis factors receptor associated signalling molecules and their role in activation of apoptosis, JNK and NF-κBAnnals of the Rheumatic Diseases, 2000
- Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell DeathCell, 1996
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- TNF-Dependent Recruitment of the Protein Kinase RIP to the TNF Receptor-1 Signaling ComplexImmunity, 1996
- FADD, a novel death domain-containing protein, interacts with the death domain of fas and initiates apoptosisCell, 1995
- A novel family of putative signal transducers associated with the cytoplasmic domain of the 75 kDa tumor necrosis factor receptorCell, 1994
- A Receptor for Tumor Necrosis Factor Defines an Unusual Family of Cellular and Viral ProteinsScience, 1990
- Molecular cloning and expression of a receptor for human tumor necrosis factorCell, 1990
- Molecular cloning and expression of the human 55 kd tumor necrosis factor receptorCell, 1990
- Selective immortalization of murine macrophages from fresh bone marrow by a raf/myc recombinant murine retrovirusNature, 1985