The effects of aging on pulmonary oxidative damage, protein nitration, and extracellular superoxide dismutase down-regulation during systemic inflammation
Authors:
- 1 January 2011
- journal article
- Published by Elsevier in Free Radical Biology and Medicine
- Vol. 50 (2) , 371-380
Abstract
No abstract availableKeywords
This publication has 63 references indexed in Scilit:
- Age-dependent vulnerability to endotoxemia is associated with reduction of anticoagulant factors activated protein C and thrombomodulinBlood, 2010
- Disruption of SM22 Promotes Inflammation After Artery Injury via Nuclear Factor κB ActivationCirculation Research, 2010
- CpG methylation attenuates Sp1 and Sp3 binding to the human extracellular superoxide dismutase promoter and regulates its cell-specific expressionFree Radical Biology and Medicine, 2010
- Protective effect of extracellular superoxide dismutase on endothelial function during agingAmerican Journal of Physiology-Heart and Circulatory Physiology, 2009
- Proteomic analysis of protein tyrosine nitration after ischemia reperfusion injury: Mitochondria as the major targetBiochimica et Biophysica Acta (BBA) - Proteins and Proteomics, 2009
- Decreased pulmonary extracellular superoxide dismutase during systemic inflammationFree Radical Biology and Medicine, 2008
- Transcription Factors Sp1 and Sp3 Regulate Expression of Human Extracellular Superoxide Dismutase in Lung FibroblastsAmerican Journal of Respiratory Cell and Molecular Biology, 2008
- The neutrophil serine protease inhibitor serpinb1 preserves lung defense functions in Pseudomonas aeruginosa infectionJournal of Experimental Medicine, 2007
- Advanced age exacerbates the pulmonary inflammatory response after lipopolysaccharide exposureCritical Care Medicine, 2007
- Definitions for Sepsis and Organ Failure and Guidelines for the Use of Innovative Therapies in SepsisChest, 1992