Redox-dependent regulation of satellite cells following aseptic muscle trauma: Implications for sports performance and nutrition
- 1 December 2020
- journal article
- review article
- Published by Elsevier in Free Radical Biology and Medicine
- Vol. 161, 125-138
Abstract
No abstract availableKeywords
Funding Information
- Hellenic Foundation for Research and Innovation
This publication has 172 references indexed in Scilit:
- Redox homeostasis: the linchpin in stem cell self-renewal and differentiationCell Death & Disease, 2013
- Wnt7a–Fzd7 signalling directly activates the Akt/mTOR anabolic growth pathway in skeletal muscleNature Cell Biology, 2011
- Prdm16 promotes stem cell maintenance in multiple tissues, partly by regulating oxidative stressNature Cell Biology, 2010
- Myoblasts from affected and non‐affected FSHD muscles exhibit morphological differentiation defectsJournal of Cellular and Molecular Medicine, 2010
- Antioxidants prevent health-promoting effects of physical exercise in humansProceedings of the National Academy of Sciences, 2009
- Redox-based regulation of signal transduction: Principles, pitfalls, and promisesFree Radical Biology and Medicine, 2008
- Tissue inhibitor of metalloproteinase-2 (TIMP-2) regulates myogenesis and β1 integrin expression in vitroExperimental Cell Research, 2008
- Inflammatory monocytes recruited after skeletal muscle injury switch into antiinflammatory macrophages to support myogenesisJournal of Experimental Medicine, 2007
- Macrophages promote muscle membrane repair and muscle fibre growth and regeneration during modified muscle loading in mice in vivoThe Journal of Physiology, 2006
- N‐acetylcysteine attenuates the decline in muscle Na+,K+‐pump activity and delays fatigue during prolonged exercise in humansThe Journal of Physiology, 2006